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MICROPORT ACADEMY CRM
DAI Boston Scientific
BIOMONITOR IIIm BIOTRONIK

ECG

ECG, practice reading and et interpreting.

1:1 Flecainide associated flutter

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Patient
67-year-old woman, hypertensive treated with flecainide for paroxysmal atrial fibrillation; palpitations with lipothymia; recording of this tracing at entry;
Trace
Very rapid tachycardia (over 200 bpm) with wide QRS (left axis, left delay); there is no typical conduction aberration pattern (right bundle branch block or left bundle branch block); atrial activation is difficult to visualize; it is therefore not possible to formally differentiate between ventricular tachycardia and 1:1 flecainide associated atrial flutter;
Trace
This second tracing was recorded after the intravenous infusion of beta-blockade; slowing of the ventricular rate, narrowing of the QRS complexes (left anterior fascicular block); the presence of long diastoles (2:1 or 3:1 conduction) allows evidencing a characteristic pattern of common atrial flutter; the first tracing corresponds to a 1:1 conducted flecainide associated atrial flutter with rate-dependent prolongation of the QRS duration (the ventricular rate on the first tracing is very similar to that of the atrial flutter rate);
Comments

The electrophysiological properties of the atrioventricular node (decremental conduction) normally allows filtering the rapid atrial activation observed during a common flutter with the onset of a protective atrioventricular conduction block (2:1, 3:1, etc.).

Exergue
A wide QRS tachycardia in a patient receiving class Ic antiarrhythmic treatment should trigger the diagnosis of 1:1 flutter. The electrical pattern of this rhythm disorder is difficult to differentiate from that of a ventricular tachycardia with very wide QRS complexes of atypical morphology and an atrial activity that is often problematic to identify.
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61
Library
Rhythm disorders
Pathology
AF, flutter, atrial tachycardia
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